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Other studies have shown that the other PD drugs acted independently to shape different microbial profiles, which strengthens the rationale for disentangling drug effects from disease signatures (Hashish and Salama, 2023)

Heres why: Short enough that IGF-1 levels wont crash dramatically

Depletion of the glutathione content of isolated liver cells by hepatotoxic drug

Mycotoxins like gliotoxin and ochratoxin A have been shown to directly stimulate mast cell degranulation and trigger a pro-inflammatory state that fuels downstream tissue damage