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These interventions aim to deliver targeted symptom relief directly within the joint space, with the principal agents including corticosteroids, hyaluronic acid (HA), and emerging biologics such as platelet-rich plasma (PRP) and cell-based products (Wehling et al., 2017

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Mucosal Immunol 13(4):637651 Wong CK, McLean BA, Baggio LL, Koehler JA, Hammoud R, Rittig N et al (2024) Central glucagon-like peptide 1 receptor activation inhibits Toll-like receptor agonist-induced inflammation

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